Millions world wide carrying gene variant that controls 'explosive' cell death linked to inflammation.
This gene could potentially explain why some people are more likely to develop inflammatory diseases and pave the way for more individualized treatment.
Cell death is the process in which the body rids itself of damaged, unwanted or diseased cells. Apoptosis is one form of cell death and is considered to be "programmed" because it's a highly regulated process. Another form of cell death is called necrosis, which is considered to be "unprogrammed" and is uncontrolled. Necroptosis is a form of cell death that mimics both apoptosis and necrosis.
"Necroptosis is distinguished from other forms of cell death by its ferocity: essentially, the cells explode, setting off an alarm that other cells in the body respond to. Whereas apoptosis usually causes an immunologically silent response, necroptotic cell death, which is gatekept by the MLKL gene, releases molecules that promote inflammation and can lead to disease."
A recent study conducted at the Walter and Eliza Hall Institute in Melbourne, Australia examined how necroptotic cell death is affected when the gene that controls it is altered.
“This is a good thing in the case of a viral infection, where necroptosis not only kills the infected cells but instructs the immune system to respond, clean things up, and start a more specific, long-lived immune response,” said Sarah Garnish, the study’s lead author. “But when necroptosis is uncontrolled or excessive, the inflammatory response can actually trigger disease.”
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